Carcinogenesis and dietary fat

Cover of: Carcinogenesis and dietary fat |

Published by Kluwer Academic Publishers in Boston .

Written in English

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Subjects:

  • Cancer -- Nutritional aspects.,
  • Lipids in nutrition.,
  • Fat -- Physiological effect.,
  • Carcinogenesis.,
  • Eicosanoids -- Physiological effect.,
  • Food -- Fat content -- Health aspects.,
  • Dietary Fats -- adverse effects.,
  • Neoplasms -- etiology.

Edition Notes

Includes bibliographies and index.

Book details

Statementedited by S. Abraham.
SeriesProstaglandins, leukotrienes, and cancer ;, PLAC6
ContributionsAbraham, S.
Classifications
LC ClassificationsRC268.45 .C37 1989
The Physical Object
Paginationxvii, 492 p. :
Number of Pages492
ID Numbers
Open LibraryOL2057929M
ISBN 10079230117X
LC Control Number88036981

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Nutritional Methodology in Dietary Fat and Cancer Research.- Effects of Dietary Fat on Eicosanoid Production in Normal Tissues.- 3. Effects of Dietary Fat on Eicosanoid Production in Normal Tissues.- 4. Eicosanoids, Their Dietary Precursors and Drugs that Modify their Production or Actions: Implications in Cancer.- Mammary Tumors.- 5.

Carcinogenesis and Dietary Fat. Editors (view affiliations) S. Abraham; Book. 40 Search within book. Front Matter. Pages i-xvii. PDF. Dietary Formulation and Feeding Regimens.

Effects of Dietary Fat on Eicosanoid Production in Normal Tissues. Front Matter. Pages PDF. Effects of Dietary Fat on Eicosanoid Production in Normal. Carcinogenesis and Dietary Fat. Editors: Abraham, S.

(Ed.) Role of Dietary Fat in Experimental Pancreatic Carcinogenesis. Pages Book Title Carcinogenesis and Dietary Fat Editors. Abraham; Series Title Prostaglandins, Leukotrienes, and Cancer.

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Free shipping for many products. Carcinogenesis and Dietary Fat (Prostaglandins, Leukotrienes, and Cancer (6)) Softcover reprint of the original 1st ed. Edition by S. Abraham (Author) ISBN ISBN Why is ISBN important. ISBN.

This bar-code number lets you verify that you're getting exactly the right version or edition of a book. Cited by: Get this from a library. Carcinogenesis and Dietary Fat. [S Abraham] -- Prostaglandins, Leukotrienes, and Cancer is a multi-volume series which focuses on an emerging area of cancer research.

InR.H. Williams first reported that elevated prostaglandin levels are. Dietary fat Carcinogenesis and dietary fat book mammary carcinogenesis. Carroll KK, Braden LM. Evidence that dietary fat has an influence on carcinogenesis comes from both epidemiological data and experiments with animals.

The experimental studies have indicated that dietary fat acts primarily as a promoter of carcinogenesis and that the effect depends on the type as well as Cited by: Carcinogenesis and Dietary Fat.

Prostaglandins, Leukotrienes, and Cancer (Book 6) Share your thoughts Complete your review. Tell readers what you thought by rating and reviewing this book.

Rate it * You Rated it *Brand: Springer US. Dietary fat accounts for approximately 50% to Carcinogenesis and dietary fat book of the nonprotein calories consumed by infants and approximately 30% of nonprotein calories consumed after age 2 years (Box ). 35 Dietary fat is ingested principally in the form of triglycerides containing the fatty acids palmitate and oleate (C and C, respectively).Dietary triglycerides of animal origin predominantly contain.

Thus, diets high in unsaturated fat appear to promote pancreatic carcinogenesis in the azaserine-treated rat while a diet high in saturated fat failed to show a similar degree of enhancement of. Preview this book» What people are The Role of Dietary Fat in Carcinogenesis. Dietary Fat and Breast Cancer.

Experimental Research on Dietary Lipids and Colon Cancer. Antioxidants Dietary Fat and Carcinogenesis. The Interrelationship between Dietary Lipids Endocrine Activity. /5(1).

3 Diet and the Mechanisms of Carcinogenesis. we should keep in mind that the important dietary determinants of cancer rates in humans may not fall readily into one of these two classes. For example, it has not been easy to decide what mechanism is most likely to be responsible for the observed effect of a high fat diet on the incidence of.

Discussion. The association between prostate carcinogenesis and high dietary fat consumption has been shown in numerous studies (1 – 10), and tissue oxidative stress is presumed to be one of the mechanisms involved (11 – 14, 16, 17).The association has not been well reproduced in animal studies (7, 18 – 21) however, and the role of oxidative stress in Cited by: Dietary fat accounts for approximately 50% to 70% of the nonprotein calories consumed by infants and approximately 30% of nonprotein calories consumed after age 2 years (Box ).

35 Dietary fat is ingested principally in the form of triglycerides containing the fatty acids palmitate and oleate (C and C, respectively). Dietary triglycerides of animal origin predominantly contain.

Dietary Fat, Gene Expression, and Carcinogenesis With Howard P. Glauert This chapter discusses the role of dietary fat on the development of 'human and experimental cancer and examines the role of gene expression as a mechanism by which dietary fat may alter the development of : Howard P.

Glauert. G.A. Boissonneault, C. Elson, and M.W. Pariza, Net Energy Effects of Dietary Fat on Chemically Induced Mammary Carcinogenesis in F Rats, J Nat Cancer Inst. PubMed Google Scholar by: Carcinogenesis and Dietary Fat.

por. Prostaglandins, Leukotrienes, and Cancer (Book 6) Comparte tus pensamientos Completa tu reseña. Cuéntales a los lectores qué opinas al calificar y reseñar este libro.

Califícalo * Lo calificaste *Brand: Springer US. This book chapter provides a comprehensive, literature-based review on dietary factors and their molecular mechanisms of prostate carcinogenesis. A large portion of our knowledge is based on epidemiological studies where dietary factors such as cancer promoting agents, including high-fat, dairy products, alcohol, and cancer-initiating Cited by: 1.

Dietary factors related to cancer development are poorly understood, although dietary practices are suspected to alter cancer risk. Suspected dietary risk factors include low fiber intake and a high intake of red meat or animal fat.

Preservatives, preparation methods, and additives (dyes, flavorings, sweeteners) may have cancer-promoting effects.

The aim of the present paper is to give a brief overview on the role of dietary fat in carcinogenesis and as possible anticancer agents. Dietary fat is an essential nutrient and important source for the essential fatty acids (FA), linoleic and α-linolenic acids, which Cited by: It also examines various diet-related initiators and promoters involved in colon carcinogenesis and the mechanism whereby dietary factors modulate colon carcinogenesis.

The concept that dietary fat and certain fibers distinct from chemical contaminants of diet and from other environmental and genetic factors are important determinants of colon Cited by: 3. In general, the evidence suggests that some types of diets and some dietary components (e.g., high fat diets or the frequent consumption of salt-cured, salt-pickled, and smoked foods) tend to increase the risk of cancer, whereas others (e.g., low fat diets or the frequent consumption of certain fruits and vegetables) tend to decrease it.

A PRELIMINARY STUDY CONCERNING THE POSSIBILITY OF DIETARY CARCINOGENESIS* R. WILLHElM, M.D. AND A. IvY, PH.D., M.D. From the Department of Clinical Science, University of Illinois College of Medicine, Chicago INTRODUCTION Gastrointestinal cancer hypothetically may arise from external carcinogens.

Plants consumed by humans contain thousands of phenolic compounds. The effects of dietary polyphenols are of great current interest due to their antioxidative and possible anticarcinogenic activities.

A popular belief is that dietary polyphenols are anticarcinogens because they are antioxidants, but direct evidence for this supposition is lacking. This chapter reviews the Cited by: Experiments to establish nutrient requirements have demonstrated that the amount of selenium required for optimum nutrition is dependent on dietary levels of fat, vitamin E, and certain heavy metals.

There is also evidence that at least the interaction between selenium and fat is an important determinant of the element's effect on cancer. When these forms ofcancerareexaminedcarefully, it isclearthat they share anumber ofcommon etiologic factors related to dietary fat, lipids, and hormones.

A human cancer is usually discovered at a point where it has formed a detectable mass. For many forms of cancer, this may require 10 to 15 years from the time when the cancer is first initiated.5/5(1). Diet Composition, Weight Control, and Breast Carcinogenesis (CHOICE) The safety and scientific validity of this study is the responsibility of the study sponsor and investigators.

Listing a study does not mean it has been. Recent studies of the effects of dietary fat on experimental carcinogenesis have probed the influence of the dietary fat source, the time during carcinogenesis when a high-fat diet is given, the interactions between fat and energy, and the potential mechanisms for the observed effects of fat on cancer.

The essential fatty acids in the fat, the degree of their unsaturation, and the Cited by: Cancer chemoprevention is a dietary or therapeutic strategy to prevent, suppress, or delay carcinogenesis either at initiation or progression level with nontoxic agents.

Future research is needed to elucidate the interactive role of tea catechins and other dietary cancer-inhibitory compounds in mammary carcinogenesis in humans. Ovarian Cancer. In a clinical study conducted on ovarian cancer patients in China, researchers found that women who drank at least one cup of green tea per day survived longer with the Cited by: 1.

For more than 50 years, it has been recognized that diet influences cancer formation both in humans and in experimental animals. In fact, early investigators successfully retarded the onset of tumors in animals by dietary manipulation. Such findings. Dietary fat quantity and composition induce changes in proliferation and membrane lipids in rat colon cells.

Annals of Nutrition and MetabolismProgress 01/01/98 to 12/31/98 Outputs We completed an experiment designed to examine the cellular interaction between dietary fat quantity and type on colon cell expression of IGF.

Specific versus non-specific effects of dietary fat on carcinogenesis. Prog Lipid Res. This review describes the specific and nonspecific effects of dietary fat on carcinogenesis. The non specific effect increases the risk through achievement a of a positive energy balance leading to obesity.

Dietary factors are believed to play an important role in mammary carcinogenesis. International correlations, case-control and cohort studies hove associated the incidence and mortality from breast cancer with high fat consumption in the form of meat, gravy and dairy products.

Most of these studies have been conducted in the developed countries. adipose tissue albumin alcohol consumption animal assessment body mass body weight breast cancer breast cancer risk calories cancer incidence cancer prevention Cancer Res carbohydrate carcinogenesis carcinogens case-control study cell cholesterol Clin cohort studies colon cancer colorectal cancer corn oil correlation diet and cancer dietary fat.

The aim of the present paper is to give a brief overview on the role of dietary fat in carcinogenesis and as possible anticancer agents. Dietary fat is an essential nutrient and important source for the essential fatty acids (FA), linoleic and α-linolenic acids, which contribute to proper growth and by: Author interviews, book reviews, editors' picks, and more.

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Apple. Android. Windows Phone Manufacturer: Informa Hralthcar,   Mechanisms of Chemical Carcinogenesis provides information pertinent to the fundamental mechanisms of chemical carcinogenesis. This book surveys the interactions of chemical carcinogens with native DNA, the activation of normal cellular sequences, and the transforming role of the activated Edition: 1.

The incidence of skin carcinomas induced by benzpyrene was studied in female Rockland strain mice given low (2 per cent.) and medium (27 per cent.) fat diets with different energy values, and low (2 per cent.) and high (61 per cent.) carbo hydrate-free fat diets of the same energy values.

As the carbohydrate-free high-fat diet stimulated carcinogenesis about as much as diets Cited by: limited suggestive evidence- high fat diet increases risk of post meno breast and lung cancer-limit suggest.

evidence that diets high in animal fat may increase the risk of bowel cancer -Cancer council supports dietary guidelines for fat: limit saturated fat and moderate fat intake-choose lean red meat and reduced fat dairy foods.

In the present work, the novel A/J Min/+ mouse model for Apc-driven colorectal cancer was used to investigate the effect of dietary heme ( μmol/g), combined with high (40 energy %) or low (10 energy %) dietary fat levels, on intestinal carcinogenesis.

At the end of the dietary intervention period (week 3–11), spontaneously developed Cited by: 3. The complexity of these relationships can be illustrated with two dietary factors: fat, and fruits and vegetables. Dietary fat provides energy at a high density with a low satiety for the calories consumed.

Consequently, as we have noted, energy availability is increased and colon carcinogenesis is promoted. Dietary fat, however, is by: A high-fat diet promotes intestinal tumorigenesis independently of obesity in a mouse model with oncogene activation, by changing the composition of the .

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